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Kruger, L

The molecular link between mitochondrial oxidative stress/DNA damage and ferroptosis in LN (223) is also poorly defined: it is unknown whether mitochondrial dysfunction directly triggers iron overload/lipid peroxidation or indirectly by suppressing antioxidant networks (e.g., Nrf2/GPX4)

The strong antioxidant properties of PQQ are reflected in its ability to neutralize free radicals, reduce oxidative stress, and help protect cells from oxidative damage It also catalyzes repeated redox reactions to a large extent

DM is characterized by disordered glucose metabolism, manifested as decreased glycogen synthesis and increased glycogenolysis