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Peptides occupy a central position in AD pathobiology: pathogenic species such as A oligomers and tau-derived fragments drive synaptic failure, mitochondrial dysfunction, and neuroinflammation, whereas several endogenous neuropeptides exert compensatory neuroprotective, trophic, or homeostatic effects

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For individuals with a deficiency, replacing B12 may help improve symptoms related to low vitamin levels

Ferroptosis regulation in ILCs is an emerging area, with implications for diseases such as asthma and fibrosis (100102)