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Thus, the dynamics of GLP-1 needs to be carefully investigated in hyperthyroid mouse models in the future, which will help to understand the glucose metabolism in patients with clinical hyperthyroidism

Chronic cerebral hypoperfusion induces microglial activation, polarized toward M1 (pro-inflammatory) phenotype, and the drug fingolimod has been shown, in an animal model, to attenuate such microglia-mediated neuroinflammation and to promote oligodendrocytogenesis by shifting microglia toward M2 subtype (217)

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alpha-ketoglutarate orchestrates macrophage activation through metabolic and epigenetic reprogramming