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Description

Nitric oxide is the primary mediator of cytotoxicity induced by GSH depletion in neuronal cells

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In a rat model of DOCA-salt hypertension, vascular mitochondrial ROS production was markedly elevated, accompanied by the activation of other ROS sources

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Paracetamol exerts a spinal, tropisetron-reversible, antinociceptive effect in an inflammatory pain model in rats

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