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The intermediates from de novo lipogenesis in the liver will disrupt mitochondrial function and produce ROS, and then cause endoplasmic reticulum (ER) stress, ultimately leading to inflammation, and the broken cells releasing cytokines will cause more cell death ( The excess free fatty acids (FFAs) and de novo lipogenesis would break the homeostasis of lipid metabolism

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Our results show that L-GSH lowered TGF-beta and IL-6

Despite visible darkening in both cases, pigmentation should not be interpreted as protection, as UV exposure can still contribute to skin aging and long-term damage, she added