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Preclinical studies have demonstrated that NCoR1 depletion, followed by treatment with rapamycin, anti-mycin-A, or metformin restored TFEB activity and LC3 levels, leading to improved clearance of M.tb [28]

The suggested mechanism could be the deactivation of hydroperoxide through the reversion of riboflavin from the reduced form (dihydroriboflavin) to the oxidised form (Fig

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A 2023 systematic review and meta-analysis published in JAMA analyzed 118 clinical trials and found that oral naltrexone was associated with significant reductions in heavy drinking compared with placebo