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Gao, H., Liu, C., Song, S
It seems in pre-clinical models that when these L-cells are exposed to perceived toxins, such as lipopolysaccharides from certain bacteria that this triggers a surge in endogenous GLP-1 which is perhaps the bodys way of using GLP-1 as an anti-inflammatory hormone or perhaps as a brake pedal on the inflammatory response in general
That is exactly what makes the rest of the story a cautionary tale rather than an obvious hoax: a plausible idea, a spectacular number, and a foundation that did not hold