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Curcumin ameliorates reserpine-induced pain-depression dyad: behavioural, biochemical, neurochemical and molecular evidences

Whereas the levels of 1,2-benzoquinone, 11-dehydro-thromboxane B2, 12-KETE, L-malic acid, N2-succinyl-L-ornithine, ornithine, argininosuccinic acid from the fecal metabolites gradually decreased as CKD progressed, chlorohydroquinone, hydroquinone, L-cystine, fecal 12-keto-tetrahydro-leukotriene B4 (12-keto-tetrahydro-LTB4), and L-cysteine levels gradually increased Integrated network analysis based on metabolomics and metagenomics data revealed that Ruminococcus bromii , fecal hydroquinone, and serum creatinine were the major contributors to this network, therefore play major roles in CKD progression

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Dysbiosis, characterized by a reduction in beneficial SCFAs and an increase in uremic toxins (IS, PCS, TMAO, and IAA), damages the intestinal barrier and elevates LPS and pro-inflammatory cytokines (TNF-, IL)