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This bidirectional effect highlights the necessity for context-specific modulation of the autophagy-ferroptosis axis

Body protective compound-157 enhances alkali-burn wound healing in vivo and promotes proliferation, migration, and angiogenesis in vitro

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In addition, nausea is believed to result mainly from central mechanisms, in which activation of GLP-1R neurons in the area postrema plays a predominant role rather than from delayed gastric emptying alone 46