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In another cellular study, miR-362-3p is shown to directly bind to and negatively regulate the expression of a disintegrin and metalloproteinase with thrombospondin motifs 1 (ADAMTS1), which suppresses VSMCs proliferation and migration in atherosclerosis

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All animal procedures were conducted in accordance with the Guidelines for Ethical Review of Animal Welfare (GB/T 358922018, China) and approved by the Experimental Animal Ethics Committee of Guizhou Medical University (No.2200040)

brasiliensis in HFD-induced obese mice were only partly dependent on STAT6, but fully dependent on IL-13 (95)