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APOL1-mediated monovalent cation transport contributes to APOL1-mediated podocytopathy in kidney disease

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And so having that little bit of GIP1 agonism on board might be more appropriate for somebody with more of a metabolically dysfunctional profile and someone like you and I whos just using it for different reasons, longevity, anti, you know, anti inflammatory, whatever we would probably need

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