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Reich N, Hlscher C

Associated insulin signaling pathways include mitogen-activated protein kinases (MAPKs), IB-kinase b (IKb)/nuclear factor B (NF-B), and mammalian target of rapamycin (mTOR)/S6 kinase [44]

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In studies of myocardial reperfusion injury, it has been found that, in the early stages of reperfusion, moderately increased ROS can activate STAT3 phosphorylation, which then translocates to the mitochondria and nucleus, thereby alleviating cytosolic calcium overload during ischemia/reperfusion (I/R) injury [259, 260]