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Bessede A, Gargaro M, Pallotta MT, et al

ROS acts as a central hub connecting ferroptosis to PCa pathogenesis, amplified by inflammatory signals and redox-sensitive transcription factors (e.g., Nrf2, HIF-1) that either directly enhance oxidative stress or indirectly modulate ferroptosis susceptibility [Figure 2A]

it is not approved for human or veterinary use, and nothing below describes or recommends any form of administration to people

Demonstrating the importance of these programs for cell fitness and survival, strategies that inhibit key NRF2 effectors or cause cysteine depletion can induce apoptosis and other forms of cancer cell death both in vitro and in vivo (60, 61)