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Our findings show that semaglutide increases the -catenin by increasing the cAMP which activates the protein kinase A (PKA) leading to the modification of -catenin enzymatically by adding phosphate groups to its Ser675 residue, which facilitates the accumulation and translocation of -catenin into the nucleus

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While the researchers found no significant differences were found in the parameters over a 12-month period, their results also showed a trend of BMI reduction and possible acetazolamide dose reduction following GLP-1 initiation

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