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10.1038/nrm3013 150

doi: 10.4049/JIMMUNOL.2000708 74 AndersonWBarahmand-Pour-WhitmanFLinsleyPSCerosalettiKBucknerJHRawlingsDJ

DefrreSLangendoncktAVVaesenSJouretMGonzlezRRet al

However, while PDZ-domain interactions may contribute to CFTR-ACE2 binding and to spike protein- or virus- dependent loss of CFTR by endosomal recycling, the responsible mechanisms for these interactions remain to be determined