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The Ferguson reflex for fetal ejection: Fetal stretch signals from the birth canal and uterine tissue activates spinal and vagal inputs to the brainstem including the solitary tract nucleus, which then sends a noradrenergic depolarizing input to hypothalamic oxytocin neurons

In hypopituitarism, GH deficiency and low cortisol due to adrenocorticotropic hormone (ACTH) deficiency result in increased insulin sensitivity and impaired gluconeogenesis, leading to recurrent hypoglycemia

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When NADPH is exogenously given at high levels (30%), INP simulations show hyper-reduction of mitochondrial complexes IIV, leading to reverse electron leakage and ROS bursts