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In parallel, AMPK can promote autophagy initiation (e.g., via ULK1 activation and mTORC1 restraint), providing a route for lipophagy and mobilization of lipid substrates that feed mitochondrial and peroxisomal oxidation (He et al., 2020)

Its metabolic functions include: Enhanced immune function Elimination of toxins Elimination of carcinogens Antioxidant cell protection Protections against ionizing radiation DNA synthesis and repair Protein synthesis Prostaglandin synthesis Amino acid transport Enzyme activation and regulation These functions can be summarized into three general categories: 1