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Briefly, therapeutic interventions for asthma primarily target oxidative stress pathways through Nrf2 activators and mitochondrial antioxidants, while addressing inflammatory cascades via JAK-STAT inhibitors, NFB modulators, and inflammasome inhibitors

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Rather than mimicking a satiety hormone, it works upstream in the central nervous system: it triggers the release of norepinephrine, dopamine, and serotonin , which suppresses appetite and modestly increases energy expenditure