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Interestingly, it has been found that this syndrome can also be caused by a mutation in the mitochondrial ribosomal protein MRPS22 with deficiencies of OXPHOS complexes I, III, and IV in fibroblast mitochondria [392]

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People with cerebrovascular disease show a significant increase in formin-like protein 2, which is responsible for amyloid and phosphorylated tau deposition and progression of AD [35]