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Laboratory studies suggest the peptide can shift gene-expression patterns toward a profile associated with younger tissue and may support the activity of cells involved in repair

Therefore, current antiinflammatory therapies in PCOS, though not explicitly designed for NET inhibition, may indirectly suppress neutrophil activation, NET formation, and subsequent tissue injuryforming a practical backdoor into the neutrophilNET therapeutic space

Many patients follow the program for several months, then reassess with their provider about continuing, adjusting, or transitioning to maintenance