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These phase I oxidative reactions metabolise 210% of paracetamol to a reactive intermediate, N -acetyl- p -benzoquinone imine (NAPQI) which mediates the hepatotoxic effect of paracetamol

Patients are educated to recognize early signs of re-emerging deficiency (increased fatigue, tingling) to prompt timely retreatment

Additionally, lipotripeptides (DICAMs) inhibit and disrupt bacterial biofilms, and GPE supports neuroprotection and regeneration through ERK and PI3K/Akt signaling activation

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