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However, the precise biophysical mechanisms and receptor-effector interactions that differ between different biased GLP-1RAs and explain their differential endocytic rates have not been fully elucidated

10.18632/aging.204555 134 ZhaoQ.LariosK.NaaldijkY.ShermanL

Functional and phenotypic analyses confirm the induction of a pancreatic endocrine fate, including positive dithizone (DTZ) staining indicative of insulin production, as well as the upregulation of key pancreatic markers such as Nestin , PDX-1 , paired box gene 4 ( PAX4 ), neurogenin 3 ( NGN3 ), Isl-1, MAFA, insulin, and glucose transporter 2 ( GLUT2 )

It's not an immediate effect, as cellular processes require time to adapt and respond