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Furthermore, the TME dictates context-specific effects

31 Together, these results indicate astrocytic GABA dysregulation as a key feature of PTSD, driven by increased MAOB- and ABAT-dependent GABA metabolism, leading us to further investigate the cellular sources of GABA within a PTSD mouse model

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Non-ceruloplasmin-bound copper (NCC) represents the redox-active free copper fraction, which is a key driver of oxidative injury and cuproptosis