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To address this question, we performed an integrated time-resolved analysis of the renal transcriptome and proteome in parallel with the renal and plasma metabolomes in control mice and mice with an induced deletion of Bmal1 in the renal tubule

Taken together, these data suggest CB0313.1 may mitigate energy metabolic dysfunction via reducing inflammatory tone and promoting mitochondrial metabolism in adipose tissue

(136) adopted a CIA mouse model and an in vitro iTFR cell culture system