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identify an unexpected metabolic underlying mechanism for this disease: the lipid chaperone fatty-acid-binding protein 5 (FABP5) aggravates psoriatic inflammation by driving keratinocytes into ferroptosis, a form of regulated cell death induced by iron-dependent lipid peroxidation (Fig

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Loren Pickart from human plasma, and since then it has been the subject of more than 200 published studies spanning wound healing, dermatology, hair biology, gene expression, and most recently, longevity research