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(II) Despite the intracellular GSH being significantly consumed in the cases stated in this review, it is difficult to completely deplete GSH via these strategies

Whether this pathway is involved in SFN induced dephosphorylation of STAT3 remains to be elucidated, but the ROS-mediated regulation of STAT3 may partially connect SFN-treatment to the strongly inhibited T H 17 cell responses on the molecular level

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Indeed, the use of these injectable substances, many of which are not approved by the U.S