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Thus, as far as mitochondrial oxidative stress homeostasis is concerned, mGSH is a vital factor maintaining mitochondrial function, controlling cell survival/death, and its depletion causes cell damage and facilitates cell death [57, 59]

Access this article Similar content being viewed by others References Ball L, Accotto G-P, Bechtold U et al (2004) Evidence for a direct link between glutathione biosynthesis and stress defense gene expression in Arabidopsis

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