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Importantly, formulation efforts emphasize stability under physiological conditions and resistance to fibril formationa limitation of earlier analogues such as pramlintide (Kruse et al

Downregulation of PCBP1/2 mediates Fe 2+ release and induces ferroptosis To explore the potential mechanism by which ACE elevates Fe 2+ levels, iron metabolism proteins associated with ACE were screened by Western blot based on proteomic results (Fig

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