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We suggest that the role of the kidney is crucial for the effect of SGLT-2i on the clinical outcomes not only because these drugs slow-down the time-dependent decline of kidney function and enhance the response to diuretics, but also because they attenuate the meal-related anti-natriuretic pressure (lowering postprandial hyperglycemia and hyperinsulinemia and preventing proximal sodium reabsorption), which would reduce the individual sensitivity to day-to-day variations in dietary sodium intake

[1,2] It appears to be potentially more influential on cellular activities than rhIGF-1 due to an apparent significant improvement in its biological activity

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In this module, the lists of differentially expressed genes and differentially changed lipids were uploaded with their log2 fold change