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A Role for the Krebs Cycle Intermediate citrate in metabolic reprogramming in innate immunity and inflammation
Research materials referenced in this article: BPC-157, TB-500, BPC-157 and TB-500 together, and MOTS-c
Its core pathological feature is excessive accumulation of adipose tissue, accompanied by abnormal proliferation, differentiation, and metabolic dysfunction of adipocytes, which in turn induce a series of complications including insulin resistance, nonalcoholic fatty liver disease, and cardiovascular diseases
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