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Upon binding to IGF-1R, the peptide initiates multiple intracellular signaling cascades, including the phosphoinositide 3-kinase (PI3K)/Akt/mTOR pathway, which regulates protein synthesis and cell growth, and the mitogen-activated protein kinase (MAPK) pathway, which controls cell proliferation and survival

These results uncovered that pressure overload impaired cardiac mitophagy, and reversed effect of semaglutide on LVH was correlative with increased cardiac mitophagy

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