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This abnormal load distribution further impairs the mechanical function of the disc, transferring excessive stress to the annulus fibrosus, leading to its damage and contributing to pain 6,26

The oxidized GSSG is then recycled by GSH reductase, sustaining a high intracellular GSH/GSSG ratio

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Current research has not identified a specific membrane receptor exclusively responsible for BPC-157's effects, suggesting the peptide may interact with various signaling systems or function through novel mechanisms not yet fully characterized