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Abstract We previously showed that oral administration of exogenous glutathione (GSH) exerted a direct and/or indirect therapeutic effect on ischemic stroke rats, but the underlying mechanisms remain elusive

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TB-500 (synthetic thymosin -4 fragment, LKKTETQ, 889 Da) shares tissue-healing indications but acts through a different mechanism (G-actin sequestration, Ac-SDKP anti-fibrosis)