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gingivalis infection induces ferroptosis in macrophages, marked by downregulation of key antioxidant defenses, such as GPX4, FSP1, and nuclear factor erythroid 2-related factor 2 (Nrf2) ( in vitro induced differentiation models, suggesting that ferroptosis in macrophages exhibits composite characteristics in terms of pathological roles

Therefore, there is an urgent need to for research on the pathogenesis of AKI and the development of new treatment options, so as to improve the treatment effects of AKI

Effect of beta-hydroxybutyrate, a cerebral function improving agent, on cerebral hypoxia, anoxia and ischemia in mice and rats

Repeat injections in the same site within 48 hours can cause localized inflammation and reduce absorption by up to 20% due to subcutaneous fibrosis