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Release 352 , 11041115 (2022)

123 Beyond agentspecific profiles, preclinical evidence clusters into mechanistic domains that map directly onto principal pathological axes of neurodegeneration

Consistently, ACC inhibition (ND630) also modestly increased histone acetylation and proliferation in the DKO cells while suppressing malonyl-CoA levels (Supplemental Figure 4D, E, F), indicating that fatty acids may act to increase histone acetylation in part through acetyl-CoA sparing from ACC consumption