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Iron overload in bone marrow-derived macrophages can upregulate SLC7A11 expression via the ROS-NRF2-antioxidant response element (ARE) axis to reduce cellular sensitivity to ferroptosis [112]

Other studies have shown that the lipogenic transcription factor carbohydrate-responsive element-binding protein (ChREBP) promotes ubiquitination and proteasomal degradation of nSREBP2 through an unexplored mechanism (28)

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