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Although both PKC and PKA have been reported to be the upstream kinase for Ser184 of GSTP in MGR3 human glioblastoma cells (51), our study indicated that only cPKC interacted with GSTP and mediated LPS-stimulated GSTP phosphorylation

Experimenting with unlicensed peptides bypasses these important safeguards and may delay diagnosis of serious underlying conditions

To detect any residual GST activity associated with the parasite extract, blank reactions control (no GSH), experimental control (no extract), and compound control (no extract plus compound at higher concentration) were evaluated