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Modulation of GST P1-1 activity by polymerization during apoptosis

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Luteolin selectively inhibits the type of O-glycosylation (Mucin-type O -glycosylation) involved in the formation of amyloid- .[ref] Animal studies show luteolin inhibits neuroinflammation by controlling microglia activation.[ref] In a mouse study of Alzheimers, luteolin protects against amyloid memory dysfunction and also increases levels of endogenous antioxidants, including Mn-SOD, Cu/Zn-SOD, and glutathione