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[32] Very few studies compare individual enantiomers with racemic lipoic acid

This paradox may be explained by the increased insulin resistance and the compensatory intra-portal hyperinsulinemia causing increased hepatic GH sensitivity that increases serum IGF-I and blunts pituitary GH secretion via the negative feedback loop

CNTF treatment was demonstrated to activate the NF-B signaling pathway, which is involved in promoting mitochondrial function and preventing neuropathy in diabetic rodents (Saleh et al., 2013)