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The AGE pathway contributes to diabetic complications through multiple mechanisms, including reduced insulin synthesis in -cells [95], neural damage via oxidative stress and inflammation [96], and direct involvement in DPN pathogenesis

When the authors subjected cells to the inflammatory cytokine IL-6, both D1 and D2 activity decreased thus resulting in low T3 and increased rT3 levels

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As such, the regulatory feedback between haem/iron sequestration and interactions with the host plays an important role in all facets if NTHi pathogenesis (Harrison et al., 2013