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Description

Data analysis was carried out using the ABI PRISM SDS 2.2.1 software (Applied Biosystems) and Microsoft Excel (Microsoft Corporation, Redmond, WA, USA)

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Blockade of voltage dependent Na+ channels and increased brain levels of GABA are possibly the result of inhibition of GABA degradative enzymes or inhibition of GABA reuptake by neuronal cells, and are though to contribute to its anticonvulsant effect

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