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In animal and cell models, GLP-1 has been shown to impact the development and/or progression of atherosclerotic plaques

in Frontiers in Endocrinology (2018) suggests this oxidative modification process may contribute to immune recognition and the development of autoimmune thyroid disease, such as Hashimotos thyroiditis and Graves disease (13,13)

BMI can over and underestimate body fat [archived link] since it doesnt distinguish fat from bone and muscle mass

Data showed that GLP-1 significantly increased PI3K, Akt, and mammalian target of rapamycin (mTOR) phosphorylation without inducing the expression of PI3K, Akt, or mTOR