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However, when PLS-DA was done, to know the variance between healthy controls and RA patients, results showed good separation (Fig

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Leptin (Anini & Brubaker, 2003), glucosedependent insulinotropic peptide (GIP) (Roberge & Brubaker, 1993), and gastrinreleasing peptide (GRP) (Persson et al., 2000), increase GLP1 release, while insulin (Lim et al., 2009) and somatostatin (Moss et al., 2012) provide negative feedback, demonstrating that GLP1 release from the gut is under multiple levels of control