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Our roGFP2 measurements confirm (i) that dynamic roGFP2 sensing (or the physiological glutathionylation/deglutathionylation of regular surface-exposed cysteinyl residues) requires a glutaredoxin activity, and (ii) that glutathionylated proteins or metabolites should be very transient redox species when GSH and glutaredoxins are present (unless they are stabilized by a kinetic uncoupling mechanism) 5,32,34,35,61,62,63,64

Although there was some variation between the studies, the overall conclusion was that sulforaphane improved aspects of the dystrophic phenotype (Sun CC

Ferroptosis antioxidant defense system mainly divided into GPX4-dependent systems (SLC7A11/GSH/GPX4 axis) and GPX4-non-dependent systems (FSP1-CoQ10-NAD(P)H axis, GCH1/BH4 axis, and DHODH/CoQH2 axis) [31, 32] (Fig