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Identification of cytochrome P450 isoforms involved in the metabolism of Syl930, a selective S1PR(1) agonist acting as a potential therapeutic agent for autoimmune encephalitis

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doi: 10.1016/S0741-8329(02)00229-X 69

The new study extends this concept to skin inflammation and proposes that increased FABP5 expression funnels these redox-active lipids toward cellular membranes while suppressing the expression of GPX4, the lipid peroxidationdetoxifying enzyme and key ferroptosis suppressor, thereby amplifying lipid peroxidation and promoting inflammatory cell recruitment (Fig