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This early work established the principle that GHK-Cu acts as a signalling molecule rather than simply a structural component, setting the stage for decades of investigation across multiple research domains

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From structural point of view, protein complexes (or aggregates) behaving as prion-like entities could represent molecular assemblies of individually folded domains or strictly cooperative folds, like amyloids, where monomers contribute toward -sheet-rich folds by layering inside the same structures, extending into fibrillar macromolecular assemblies (amyloid fibers)