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Both influence mitochondrial function, inflammatory tone, and the molecular machinery that governs neuronal survival and repair, engaging the same PI3K-Akt signaling pathway established earlier, now operating within neurons rather than peripheral tissue

The mechanism of action involved inhibition of sphingosine kinase 1 (SphK1), a sphingosine-1-phosphate (S1P)-producing enzyme, along with STAT3 and NF-Bp65, which are involved in chemokine production [91]

By selectively inhibiting nicotinamide N-methyltransferase (NNMT), 5-Amino-1MQ helps preserve intracellular NAD+ levels and redirect cellular energy metabolism toward enhanced fat oxidation and mitochondrial efficiency

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